August 15, 2006 For Immediate Release
Sherry Rust, Acting Public Information Officer
Department of Livestock
301 N. Roberts
PO Box 202001
Helena, MT 59620-2001
406-444-9321
Fax 406-444-1929
srust@mt.gov
Equine WNV now confirmed in five Montana counties
Horses in Cascade, Glacier, Phillips, and Yellowstone Counties as
well as a second horse in Flathead County have been confirmed with
West Nile Virus (WNV), for a total of six equine WNV cases this
season, according to Montana Assistant State Veterinarian Dr. Jeanne
Rankin.
The first confirmed case, a 2-year old stallion was not vaccinated
and was euthanized. Typically approximately one third of clinically
affected horses die. Since the first confirmed equine WNV case in 2002
(134 confirmed cases), the total number of infected horses in Montana
is 354 with118 deaths and 333 unvaccinated horses. There have been 10
WNV cases confirmed in 2005 and 11 in 2004. The worst year was 2003
with 193 confirmed cases with the last reported case in late October.
The age range of animals infected this year is from a 1½-year-old
filly to a seven-year-old stallion, which shows the animal does not
have to be older or debilitated to become infected with WNV.
"Historically this is about the time of year when the first
cases of WNV appear," Dr. Rankin explained, "We have had six
cases reported so far this year and we can expect to see this run
through September and into October."
"We highly recommend that horse owners talk with their
veterinarians about their horses’ risk of getting West Nile Virus
and the use of vaccines. It is not too late to vaccinate against West
Nile Virus for this year," Dr. Rankin stressed. "With the
WNV season possible into late October, there is still time to complete
the recommended two initial vaccinations which are given three to six
weeks apart."
Effective mosquito control and vaccination are the best safety
precautions for your animals against the disease. Watering troughs
should be cleaned thoroughly and regularly. A variety of water
treatment solutions are available that contain natural bacteria that,
when put into the water, will kill fly and mosquito larvae, but is
nontoxic to animals. For further information contact Greg Johnson,
Entomologist at MSU, or your local county extension agent.
Clinical signs of West Nile Virus in horses include loss of
appetite and depression, in addition to any combination of weakness or
paralysis of hind limbs, muzzle twitching impaired vision,
incoordination, head pressing, aimless wandering, convulsions,
inability to swallow, circling, hyperexcitability, or coma.
More information about West Nile Virus in horses is available from
the Department of Livestock Web site at www.mt.gov/liv.
For information regarding the disease in humans visit www.dphhs.mt.gov
Montana
has equine shipped semen regulations that require all semen shipped
into Montana must have a negative EVA & EIA test current within 6
months of shipping date and a general health inspection. The
permitting process allows the State Veterinarian's Office to verify
the requirements have been met. The permit application is
available by either phoning our office at (406) 444-2043 or visiting
our website at www.mt.gov/liv in
the forms section of Animal Health. The permitting process and
import requirements allow mare owners in Montana a safety net of
knowing that a breeding stallion is not a carrier at that time.
Many mare owners are not aware of the potential risk of spreading of
disease through shipped semen. EVA is not strictly passed
through reproductive means however. More information is
available regarding this and other diseases.
Last
week, the American Quarter Horse Association released the following
Media Advisory regarding the confirmation of an outbreak of equine
arteritis virus (EVA) infection on a American Quarter Horse breeding
farm in New Mexico. A number of horse owners have expressed
concern that movement restrictions will be placed on horses moving into
or out of New Mexico as a result of the announcement. Can you let
me know if your state is considering restrictions or have placed
restrictions as a result of the EVA outbreak reported in New Mexico.
I'd like also to take the opportunity to remind you that a great deal of
information is available about controlling and preventing this
infection. The American Horse Council developed an effective
protocol for breeding susceptible mares to EAV carrier stallions, which
has been used successfully by many breeders. In addition, a
Uniform Methods and Rules for addressing and preventing EVA outbreaks is
available from Dr. Tim Cordes at USDA. Dr. Cordes also has a very
informative video available that provides excellent information on EVA
and how to prevent its spread. And of course, if I can be of any
assistance in answering questions, please don't hesitate to contact me.
Sincerely,
Amy
Amy W. Mann
Director
Health and Regulatory Affairs
American Horse Council
1616 H Street, NW, 7th Floor
Washington, DC 20006
703-981-9745 (cell)
202-296-4031 (AHC Office)
202-296-1970 (AHC fax)
website: www.horsecouncil.org
Media
Advisory
June
30, 2006
For
Immediate Release
Equine
Viral Arteritis (EVA) Outbreak Reported in New
Mexico
On
Monday,
June 26, 2006, the Office International des Epizooties (O.I.E.)
Reference Laboratory for Equine Viral Arteritis at the University of
Kentucky College of Agriculture's Maxwell H. Gluck Equine Research
Center, confirmed an outbreak of equine arteritis virus infection
involving fetal losses among mares on an American Quarter Horse breeding
farm in New Mexico.
This
was based on the widespread prevalence of high antibody levels to the
virus in both mares and stallions, plus virus isolation from the semen
of two stallions. On the same day, the outbreak was reported to the New
Mexico Livestock Board in Albuquerque,
New Mexico, which is now investigating the potential for spread of the
infection to other premises.
The
EVA Reference Laboratory is interested in receiving samples from
suspected clinical cases of EVA or from animals very recently exposed to
semen from either of the virus-shedding stallions. Veterinarians are
requested to contact the Gluck
Center at (859) 257-4757 before submitting samples.
For
more information about this outbreak or about EVA, the following
resources are available:
What is EVA / How is it diagnosed?
http://www.xcodesign.com/aaep/displayArticles.cfm?ID=285
AAEP Guidelines for breeding a mare to an equine arteritis
virus-shedding stallion:
http://www.xcodesign.com/aaep/displayArticles.cfm?ID=36
TEXAS
COMPANIES CLONING CUTTING HORSES---ViaGen Inc. of
Austin
,
Texas
, and Encore Genetics Ltd. of
Weatherford
,
Texas
, last week announced the cloning of two champion cutting horses at a
charge of $150,000 each. The companies expect to commercially clone as
many as 30 more horses during the next 12 months at a rate of $150,000
for the first clone and $90,000 for the second clone (copy) of the same
animal.
The cloned horses, both mares, were born to recipient mares – Feb. 19
and March 9 – on Royal Vista Southwest Farms near
Purcell
,
Okla.
One of the foals is the clone of a 26-year-old champion cutting horse
with lifetime earnings of $381,764.
So
far, cloned cutting horses have not been banned from competing in
sanctioned competitions, but other horse organizations and oversight
groups already have announced bans on cloned horses racing or being
listed in purebred registries. A bill working its way through the
Oklahoma
legislature would ban cloned horses from competing in races sanctioned
by the Oklahoma Horse Racing Commission.
MONTANA REQUIREMENTS FOR EQUINE SHIPPED SEMEN
Horse owners have many choices available these days for breeding
options. With the availability of shipped cooled semen and frozen semen
for certain breeds, stallions across the nation and internationally are
now accessible for breeding to mares.
Disease prevention is a top priority and the Montana Department of
Livestock, Animal Health Division is responsible for regulating import
requirements for live animal shipments, semen, and biologics for the
state.
For breeds allowing shipped cooled semen, the Montana import
requirements for stallions include an Equine Infectious Anemia (EIA) test
current within the past 6 months, an Equine Viral Arteritis (EVA) test
current within the past 6 months, and a general health inspection current
within the past 6 months. A permit issued from the Department of Livestock
is also required. These regulations must be met prior to the shipment of
semen.
Equine Infectious Anemia, sometimes referred to as Swamp Fever, is an
infectious and potentially fatal viral disease. There is no vaccine or
treatment available for the disease. Primarily transmitted by insects, the
animal can be acutely or chronically infected. Animals in the acute stage
may develop severe signs and die within 2 to 3 weeks. This may be the most
difficult stage to diagnose as signs appear rapidly and often only an
elevated body temperature is noted. One-fifth of a teaspoon of blood from
a horse with acute EIA contains enough virus to infect 1 million horses.
Chronically infected horses may develop recurring clinical signs such as
fever, petechial (blood spots on the mucous membranes) hemorrhages,
depression, weight loss, dependent edema, anemia, or irregular heartbeat.
Equids testing positive have very few options, with most being euthanized.
Equine Viral Arteritis is an acute, contagious respiratory and abortion
disease characterized by fever; depression; dependent edema especially of
the limbs, scrotum, and prepuce in the stallion; conjunctivitis; nasal
discharge; and infrequently, death in young foals. Outbreaks are not
always characterized by clinical signs, the majority of cases are
asymptomatic. The virus can be transmitted venereally by the acutely
infected mare or the acutely or chronically infected carrier stallion.
Mares can be infected venereally either by natural service or artificial
insemination with infective semen, causing abortion or passing the disease
on to the unborn foal, which consequently dies. Carrier stallions are
viral reservoirs with the virus being shed constantly in the semen. The
infection rate by a carrier stallion is up to 85% of the susceptible mares
to which they are bred. Acutely affected stallions may suffer from a
period of short-term subfertility.
In an effort to facilitate shipments of semen without compromising the
integrity of disease control programs and allowing for the limited
time-frame semen is shipped, the Animal Health Division has created an
Annual Equine Semen Import Permit. The permit is a single sheet
application and is completed and returned back to the Animal Health
Division prior to any semen needing to be shipped. Copies of the current
EIA and EVA tests along with verification of the most current health
inspection must accompany the application. Upon receipt of the application
and supporting paperwork, a permit number is issued and a copy returned
back to the stallion manager. A copy of the permit is required to be
included with each shipment of semen. The permit is applicable per
stallion so each individual animal must have a permit. There is no charge
for the application or permit.
You may phone the Montana State Veterinarian’s Office at (406)
444-2043 for additional information or to request an application, or visit
our website at: www.mt.gov/liv/animalhealth/index.asp
March 17, 2005
Horse Travel Requires Permits and More
(HELENA) -- The amount of horse-related
travel into and around Montana increases in the spring, and the Montana
Department of Livestock (DOL) has guidelines to ensure the health, safety
and ownership of all animals involved. The guidelines apply to
horses coming into the state, leaving the state, and traveling from county
to county within Montana.
Several items are required before a person
can bring a horse into the state, according to Montana State Veterinarian
Dr. Tom Linfield, and they have certain time requirements attached to
them.
A current certificate of veterinary
inspection (CVI), also known as a “health certificate,” issued by an
accredited veterinarian in the state of origin is needed. To be a current
CVI, it must have been issued within 10 days of the veterinary exam and is
only valid for the 30 days following that exam before bringing a horse
into Montana.
A negative equine infectious anemia (EIA)
test that has been performed in the past 12 months is also required.
The negative EIA test results, test date, and the name of the laboratory
conducting the EIA test must be included on the CVI.
A Montana import permit, issued by the DOL,
is required and can be obtained within 10 days prior to transporting a
horse into Montana by calling the DOL at 406-444-2976. This is a
24-hour service.
In addition, a brand inspection is
required for horses entering Montana, as well as those traveling within
Montana. If the state of origin does not have a brand inspection
available, one may be obtained upon arrival in Montana by contacting the
local sheriff’s office for referral to a brand inspector.
For complete brand laws and requirements,
and a list of brand inspectors, horse owners can call the DOL’s Brands
Enforcement Division at 406-444-2045. The information is also
available on the DOL web page at www.mt.gov/liv.
For Montanans leaving the state with a
horse and returning, a CVI is needed for the horse. This certificate
must be issued by an accredited Montana veterinarian and is valid for 30
days to one destination. A current negative EIA test, a re-entry
permit, and a lifetime brand inspection are also required. A
lifetime brand inspection is valid for crossing state lines and is
recognized by most states. The annual brand inspection is valid for
crossing county lines within the state only. An annual re-entry
permit is also available for those taking multiple trips outside the state
each year. The annual permit is valid for a calendar year and
expires on Dec. 31.
Horse
owners should always check the brand and health requirements for the
states they will be traveling to prior to departure. “During
certain times of the year, there may be disease outbreaks, either in
Montana or destination states,” Dr. Linfield said. “If travel
includes one or more states where disease is occurring, additional
restrictions may apply to enter those states and for return to Montana.”
In addition, some states require a negative EIA test within 6 months,
rather than within 12 months.
For residents and horses traveling within
the state, a brand inspection is required to cross county lines. The
inspection may be either a trip permit, an annual inspection that expires
12 months from the date it is issued, or a lifetime brand inspection that
is valid for the entire time the listed owner retains ownership of the
horse.
For imported horses, a current
negative EIA, or coggins test, is required because of the severity of the
disease and because the disease is contagious. EIA, also known as
Swamp fever, is a viral disease, most often transmitted by biting flies,
such as horse flies or deer flies. The disease is characterized by
intermittent fever, depression, progressive weakness, weight loss, edema,
and progressive or transitory anemia, as well as occasional neurological
manifestations. The virus causes a lifetime infection in horses and
there is no cure for EIA.
“It is also recommended that a buyer
obtain a recent negative EIA test on any horse purchased,” Dr. Linfield
added. An EIA test is not required on change of ownership within the
state, but will help prevent inadvertent spread of the disease. In
addition, some shows, events or sales within Montana may require that
participating horses have current negative EIA tests.
For more information contact:
_____________________________
Karen R. Cooper, APR
Public Information Officer
Montana Department of Livestock
PO Box 202001
Helena, MT 59620-2001
406-444-9431
406-444-4316 fax
kcooper@mt.gov
*** please note new email address
for
your records ***
_______________________
COLORADO
STATE UNIVERSITY VETERINARIANS REPORT UNUSUAL RISE IN CASES OF PIGEON
FEVER AMONG COLORADO HORSES
FORT COLLINS - Equine veterinarians at Colorado State University's
James L. Voss Veterinary Teaching Hospital report a serious increase in
the number of cases of pigeon fever they have treated since early fall and
warn horse owners to be alert for signs of the highly contagious disease.
Seventy-six cases from Colorado's Front Range have been confirmed by the
Colorado State University Veterinary Diagnostic Laboratory since early
fall, more than six times the number of cases from last year's total of 12
confirmed cases and far above the seven confirmed cases in 2000."What
was once considered a disease of California horses is now a growing
problem for the Colorado equine population," said Andrea Torres,
veterinarian and microbiology resident who conducted a study of the
disease in Colorado in 2000-2001. "The increased number of confirmed
cases may be due to a more educated horse-owning public and/or to more
veterinarians being aware of the disease and testing for it."Torres
and other veterinarians at the hospital point out that the signs of pigeon
fever can also initially resemble those of other diseases such as
strangles. Sometimes the only initial signs are lameness and a reluctance
to move.Pigeon fever, also called pigeon breast, breastbone fever, false
strangles, dryland strangles or dryland distemper, is caused by
Corynebacterium pseudotuberculosis and is found worldwide. It can strike a
horse of any age, sex or breed, but usually attacks young adult animals.
There is a low incidence in foals.It has also been diagnosed in cattle,
and a similar disease affects sheep and goats. The disease is not
transmissible to humans (Note: Information from Utah State University and
Dr Bowen, a veterinarian from KY said it can be transmissible to humans,
but is rare and only in patients with suppressed immune systems, such as
an AIDs patient. It’s still is good to wear gloves or wash your
hands after being close to the lesions). Humans can carry the
infectious agent on shoes, clothing, hands or barn tools and transfer it
to another animal.Clinical signs include lameness, fever, lethargy and
weight loss and usually is accompanied by very deep abscesses and multiple
sores along the chest, midline and groin area and, sometimes, the back.
Abscesses also can develop internally.The disease is called pigeon fever
because infected animals often develop abscesses in their pectoral
muscles, which swell and resemble a pigeon's chest. Although the disease
is considered seasonal, with most cases occurring in early fall, a number
of cases have been confirmed during winter months and other times of the
year as well.The causative bacteria live in the soil and can enter the
animal's body through wounds, broken skin or through mucous membranes.
Additionally, some researchers believe pigeon fever may be transmitted by
flies.The disease occurs in three forms: external abscesses, internal
abscesses and limb infection, also known as ulcerative lymphangitis. The
most common forms are external abscess and lymphangitis, with the
prognosis of a full recovery being generally good. Internal abscesses are
much more difficult to treat."Because this disease is so highly
contagious, it is very important that veterinarians accurately diagnose
these cases to tailor treatment and control," said Torres."Horse
owners should be aware of the clinical signs and understand that
veterinary care must be timely. Infected horses should be isolated, the
abscesses properly treated and the drainage properly disposed of. The area
where the infected horse is kept must be properly cleaned and completely
disinfected because this is a very hardy bacterium. Pest control is
extremely important"As a service to horse owners, Colorado State's
equine veterinarians have created a fact sheet on pigeon fever (attached)
that is posted on the Colorado State University website at
www.cvmbs.colostate.edu/vth and www.colostate.edu on the news page.
FACT
SHEET: PIGEON FEVER IN EQUINES
Common
Names:
Pigeon fever, pigeon breast, breastbone fever, dryland distemper, dryland
strangles, false strangles, false distemperGeographic Incidence: Endemic
to California, but now found in most Western states in the U.S.Seasonal:
Usually appears in late fall but can appear sporadically at any time of
year.
Cause: Corynebacterium pseudotuberculosis
Vaccine: None at this time.
Reservoirs and mode of transmission: Can live in the soil and enter
the horse's body through wounds or broken skin and through mucous
membranes.
May possibly be transmitted by flies, including the common housefly and
horn flies.
Disease is usually highly contagious and can easily infect multiple horses
on the premises.
Bacterium in the pus draining from abscesses on infected horses can
survive from one to 55 days in the environment. It has also been shown to
survive from one to eight days on surface contaminants and from seven to
55 days within feces, hay, straw or wood shavings.
Lower temperatures prolong the survival time.
Clinical signs: Early signs can include lameness, fever, lethargy,
depression and weight loss.
Infections can range from mild, small, localized abscesses to a severe
disease with multiple massive abscesses containing liters of liquid,
tan-colored pus.
External, deep abscesses, swelling and multiple sores develop along the
chest, midline and groin area, and, occasionally, on the back.
Incubation period: Horses may become infected but not develop
abscesses for weeks.Animals affected:The disease usually manifests in
younger horses, but can occur in any age, sex, and breed.
A different biotype of the organism is responsible for a chronic
contagious disease of sheet and goats, Caseous lymphadenitis, or CL.
Either biotype can occur in cattle.
Disease forms: Generally 3 types: external abscesses, internal
abscesses or limb infection (ulcerative lymphangitis).
The ulcerative lymphangitis is the most common form worldwide and rarely
involves more than one leg at a time. Usually, multiple small, draining
sores develop above the fetlock.
The most common form of the disease in the United States is external
abscessation, which often form deep in the muscles and can be very large.
Usually they appear in the pectoral region, the ventral abdomen and the
groin area. After spontaneous rupture, or lancing, the wound will exude
liquid, light tan-colored, malodorous pus.
Internal abscesses can occur and are very difficult to treat
Diagnosis: Your veterinarian can easily collect a sample for
culture at a diagnostic laboratory. It is important to isolate the
bacterium to get a definitive diagnosis since pigeon fever can
superficially resemble other diseases.
Treatment:
Hot packs or poultices should be applied to abscesses to encourage
opening. Open abscesses should be drained and regularly flushed with
saline.
Surgical or deep lancing may be required, depending on the depth of the
abscess or the thickness of the capsule, and should be done by your
veterinarian.
Ultrasound can aid in locating deep abscesses so that drainage can be
accomplished.
External abscesses can be cleaned with a 0.1 percent povidone-iodine
solution
Antiseptic soaked gauze may be packed into the open wound
A nonsteroidal anti-inflammatory drug such as phenylbutazone can be used
to control swelling and pain
Antibiotics are controversial. Their use in these cases has sometimes been
associated with chronic abscessation and, if inadequately used, may
contribute to abscesses, according to one study.
The most commonly used antibiotic for the treatment of this condition is
procaine penicillin G, administered intramuscularly, or trimethoprim-sulfa.
In the case of internal abscesses, prolonged penicillin therapy is
necessary
Care required: Buckets or other containers should be used to
collect pus from draining abscesses and this infectious material should be
disposed of properly.
Consistent and careful disposal of infected bedding, hay, straw or other
material used in the stall is vitally important.
Thoroughly clean and disinfect stalls, paddocks, all utensils and tack.
Pest control for insects is also very important.
Recovery time: Usually anywhere from two weeks to 77 days.
Prognosis: Usually good with complete recovery, although some
horses may experience recurrence.
From: "Linfield, Dr. Thomas" <tlinfield@state.mt.us>
Sent: Thursday, July 08, 2004 4:58 PM
> *******************************************
Stomatitis in equines in Colorado
> ---------------------------------------
> On 2 Jul 2004, an equine premise in Colorado met the index case
definition
> for vesicular stomatitis (VS). Later on the same day, a 2nd equine
premise
> met the subsequent case definition for VS. The affected premises have
been
> placed under quarantine.
> VS is a viral disease which primarily affects horses, cattle, and
swine.
The
> virus that causes VS has a wide host-range. VS also occasionally
affects
> sheep and goats. In affected livestock, the VS virus causes
blister-like
> lesions to form in the mouth, on the dental pad, tongue, lips,
nostrils,
> hooves, and teats. These blisters swell and break, leaving raw tissue
that
> is so painful that infected animals generally refuse to eat and
drink.
They
> also generally show signs of lameness. Severe weight loss usually
follows,
> and in dairy cows, a severe drop in milk production commonly occurs.
> Affected dairy cattle can appear to be normal and will continue to
consume
> about half of their normal feed-intake.
> The need for increased surveillance and reporting should be
communicated
to
> all facilities, and personnel, of the animal disease monitoring
network.
> Early detection remains the key to minimizing the impact of such a
disease
> introduction.
> Please forward this information to your federal, state, and industry
> counterparts as necessary.
> For additional information on VS, please refer to the following APHIS
> web-page:
> <http://www.aphis.usda.gov/lpa/issues/vs/vs.html>
> If you have any questions about this situation, please feel free to
call
the
> Emergency Management Staff at 301-734-8073.
>
> --
> ProMED-mail
> <promed@promedmail.org>
>
> [see also:
> Vesicular stomatitis, equine, bovine - USA (TX, NM) 20040701.1754
Vesicular
> stomatitis, equine - USA (TX, NM) 20040619.1640 Vesicular stomatitis,
equine
> - USA (NM) 20040608.1538 Vesicular stomatitis - USA (TX) (04)
20040529.1466
> Vesicular stomatitis - USA (TX) (03): OIE 20040521.1367 Vesicular
stomatitis
> - USA (TX) (02) 20040520.1354 Vesicular stomatitis - USA (TX)
20040520.1350]
> ....................tg/msp/lm
>
> *##########################################################*
> ProMED-mail makes every effort to verify the reports
that are posted,
> but the accuracy and completeness of
the information, and of any
> statements or opinions based thereon, are not
guaranteed. The reader
> assumes all risks in using information posted or archived by
ProMED-mail.
> ISID and its associated service providers
shall not be held
> responsible for errors or omissions or held liable for
any damages
> incurred as a result of use or reliance upon posted or archived
material.
> ************************************************************
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HC/HERDA Update; Owners of
Carrier Stallions Notified
|
|
|
|
Approximately 100 Quarter Horse stallion owners have received,
or will receive, a telephone message from Ann Rashmir, DVM, MS,
Dipl. ACVS, associate professor of surgery and head of the
Hyperelastosis Cutis (HC) Research Program at Mississippi State
University, that contains unwelcome news. The message is that the
stallion is a carrier of the recessive gene that causes HC, also
known as hereditary equine regional dermal asthenia (HERDA).
Being a recipient of that information, says Steven Hanson, a
Billings, Mont., attorney as well as a cutting horse exhibitor and
breeder, just might serve to place a special responsibility on the
stallion owner to reveal to mare owners that the stallion is a
carrier.
Rashmir says she has no immediate plans to make the names of
the known carrier stallions public.
"I think it is only fair that the owners should be
informed first," she said. "They shouldn't have to read
about it in a publication. My hope is that they will then make
this information available to persons planning to breed mares to
their stallion or stallions."
The stallions known to be carriers are those which have sired
an HC-affected offspring. Siring even one HC offspring is proof
that the stallion is a carrier, says Rashmir.
Rashmir and Nena Winand, DVM, PhD, a geneticist and assistant
professor in the Department of Molecular Medicine at Cornell
University, announced earlier that 100% of the horses they have
studied which are afflicted with HC trace through both sire and
dam to Poco Bueno or his immediate relatives--Poco Bueno's sire,
King, and Poco Bueno's full brother, Old Granddad. Rashmir and
Winand are collaborators on HC research.
What is HC?
When a horse has HC, there is a lack of adhesion within
the dermis, the deep layer of skin, due to a collagen defect.
Collagen serves as a form of glue that holds the skin layers
together. In horses with HC, the "glue" is inferior and
the skin layers separate. When the horse is ridden under saddle or
suffers trauma to the skin, the outer layer often splits or
separates from the deeper layer, or it can tear off completely. It
rarely heals without disfiguring scars. New damaged areas arise
continuously, sometimes without obvious trauma. Click here
to see images of HC/HERDA.
The condition has manifested itself more frequently in cutting
horses, but other disciplines are not immune, with cases showing
up in reiners and pleasure horses which trace back to Poco Bueno.
Because the condition is caused by a recessive gene, it means
that both sire and dam must possess the gene before an offspring
of the two will be afflicted. Even when both sire and dam possess
the gene, the disease will not necessarily be manifested. Under
the laws of genetics, if a carrier is bred to a carrier, 25% of
the offspring will have HC, 25% will not be afflicted or be
carriers (they'll be genetically normal), and 50% will become
carriers but not be afflicted.
One of the reasons that HC rears its head more often in cutting
horse circles than any other, it is theorized, is that one of the
most popular lines traces back to Poco Bueno through Doc O'Lena
and Dry Doc and their dam, Poco Lena, a daughter of Poco Bueno.
Both Doc O'Lena and Dry Doc are proven carriers of the recessive
HC gene. Through the years, cutting horse breeders have tended to
utilize the Doc O'Lena and Dry Doc bloodlines on both sides of the
pedigree--breeding cousins to cousins. In essence, they have
created a gene pool of carriers.
"It isn't the horse's fault," says Rashmir. "The
problem revolves around the way we have bred them. Carriers have
been bred to carriers. Breeders have a responsibility to avoid
doing this if they know that stallion and mare are indeed
carriers."
This does not mean, she says, that one should avoid the Poco
Bueno bloodline when establishing a breeding program.
"If you owned Poco Bueno and knew that he had a genetic
flaw, would you have stopped using him as a breeding
stallion?" she asked rhetorically. "Not at all. He was a
wonderful athlete and a great progenitor, but you would have, or
should have, worked around the problem by avoiding inbreeding.
It's the same with many of his descendants. They are great
athletes and even if they are carriers, it has no effect on their
performance capability. Simply having the recessive gene produces
no harm to that individual. The problem arises when carrier is
bred to carrier, setting the stage for HC to be manifested."
The American Quarter Horse Association (AQHA) is funding
research at the University of California, Davis, aimed at finding
a genetic marker for the disease. Researchers say they are two to
three years away from being able to identify the recessive gene.
AQHA also has funded research at Mississippi State where
Rashmir and associates are attempting to establish a protocol for
dealing with afflicted horses. While the disease is a death
sentence for many, Rashmir said, there has been some progress in
developing ways to mitigate the effects of HC. Some afflicted
horses, she said, have been able to perform in at least a limited
manner.
Owners of Carrier Stallions
What are the legal ramifications when a stallion owner knows that
his horse is a carrier? Is he/she bound to disclose that
information to mare owners?
We asked attorney Hanson to respond to that question. Here, in
part, is what he had to say:
"Because apparently there is no method yet developed to
test through tissue biopsy for carrier status in breeding stock,
at present the only known method of determining whether a mare or
stallion is a carrier, if not themselves affected, is to confirm
the existence of the condition in affected offspring. The
production of an affected offspring means that both sire and dam
are carriers.
"That begs the question of whether the stallion owner
should make disclosure once he has knowledge of his sire's carrier
status," continued Hanson. "If a mare owner has a known
carrier, or a mare with known carrier ancestors, he can avoid
producing an affected foal by mating the mare with a non-carrier
stallion; but if the mare owner is unaware of a prospective sire's
carrier status, and the owner of the stallion does not disclose
the stallion's genetic flaw, does the mare owner have a legally
enforceable cause of action against the stallion owner in the
event the resulting offspring is affected with HC?
"Normally, saying nothing stands a better chance of
avoiding legal liability than saying the wrong thing; i.e.,
speaking falsely or in a manner that misleads," added Hanson.
"When dealing with fraud or misrepresentation, however,
silence may be construed as actionable.
"Fraud and misrepresentation, as causes of action against
the speaker, are usually associated with contract cases where the
damaged party was either intentionally or negligently induced to
enter into an agreement and then later discovered, to his
detriment, that the other party had misrepresented or falsely
stated a material fact upon which the damaged party had
justifiably relied," Hanson explained. "The other party
could even be held liable for damages, whether or not he intended
to actually cause harm to the innocent party, if by saying nothing
he covered up a fact that is construed as information that would
be material to the unsuspecting party in making a breeding
decision.
"The courts in such cases give consideration to numerous
factors, or elements of proof, and of course each case will be
decided on its own merits," he added. "In general, it
may be said, however, that failure to disclose a known defect in
the quality of the goods which are the subject of the contract
could indeed be a basis for an award of damages against the silent
non-disclosing party to the contract.
"In the end, even though it may have an adverse impact in
the form of reduced breeding fee sales, owners of stallions which
are known HC carriers may be placing themselves in legal jeopardy
if they fail to make disclosure of the existence of the genetic
defect in their breeding sires," Hanson stated. "The
duty to disclose may also extend to sellers of young horses which
have not exhibited the outward manifestations of HC, but which are
the offspring of known carriers."
Hanson has personal experience with HC, although inadvertently.
It is a story that is being told here because it demonstrates the
insidious manner in which HC can surface.
Hanson purchased a promising son of the late Shorty
Lena--tracing back to Poco Bueno through Doc O'Lena and Poco Lena.
Wondering if the young horse was stallion material, Hanson offered
to breed the stallion, free of charge, to a mare owned by a
friend. At the time, Hanson and the mare owner knew little about
HC. The mare's granddam, traced directly to Poco Bueno through a
grandparent and a great-grandparent.
The mare, bred to Hanson's stallion, produced what appeared to
be a quality filly. Before reaching one year of age, however, she
manifested clinical signs of HC. A biopsy was sent to Mississippi
State University. The diagnosis was hyperelastosis cutis.
Hanson did not face any major decisions as to how to handle the
situation because, before learning that the stallion was a
carrier, he had decided that standing a stud wasn't for him and
the horse was gelded. The mare owner had not paid a breeding fee,
so there were no damages from that aspect.
Hanson concluded his remarks on legal obligation involving HC
carriers thusly: "The duty to disclose, or lack thereof,
presents a moral as well as a legal dilemma. Ethicists may argue
the question at length, but in the final analysis, each owner of a
known carrier stallion will settle that debate for himself in his
own mental wrestling match.
"As to the legal question, persons suffering damage in
consequence of misrepresentation by silence are urged to seek the
advice of an attorney who can render an opinion after
consideration of all the facts and the law of the state which has
jurisdiction over the parties and the subject matter of the
breeding contract," concluded Hanson.
While there are approximately 100 known carrier stallions, they
are only the tip of the iceberg, said both Rashmir and Winand.
They fear that there are a great many others which have either
sired affected offspring or which have the potential to do so.
Cutting horse trainer James Johnson of Casper, Wyo., purchased
a quality two-year-old prospect from a breeder. The horse's
lineage traced back to Poco Bueno through Doc O'Lena. Johnson took
the horse to his ranch and turned it out in a pasture with some
other horses. One day he noticed the colt had what appeared to be
a slight tear in the skin on his neck. Johnson surmised that a
young Longhorn bull in the same pasture might have been
responsible. The wound was treated, but it didn't heal. Later, it
appeared to heal, but lesions opened in other parts of the colt's
body. Veterinarians were called in and specialists were consulted.
Although no biopsy was taken, the conclusion was that the young
horse was afflicted with hyperelastosis cutis.
"It was awful," Johnson said. "He just never
healed."
The veterinarians said there was nothing further they could do
and the youngster was put down.
The next question is this: Will knowing that a stallion is a
carrier dissuade mare owners from breeding to him when the mare
might also be a carrier? The answer is sometimes yes, sometimes
no. One breeder, who requested anonymity, said he is willing to
take his chances breeding carrier to carrier because the
bloodlines with which he is involved are producing winners in the
cutting pen. He has the financial means to take the risk.
There is another side to the coin. One breeder who feels she
has suffered a damaging financial loss due to HC is Cindy Lyles of
Haslet, Texas. She bred her mare and did an embryo transfer. When
flushed, the mare produced two viable embryos and each was
implanted in a surrogate mare. Two foals were born. Both of them
demonstrated outward signs of HC early in their lives. Further
analyses, including biopsies, proved they were afflicted. Lyles
has donated the two colts to the HC research herd at Mississippi
State University.
The woman estimates that she has invested $17,000 in the
breeding fee, embryo transfers, and veterinary bills. Both sides
of the pedigree trace back to Poco Bueno through Doc O'Lena.
Until a genetic test is established that will determine carrier
status, the horse owner is left with guesswork in many cases when
Poco Bueno bloodlines are involved. As an assist, Rashmir has
offered to analyze pedigrees and provide information concerning
the odds of a particular mating producing a foal with HC. The
charge for an eight-generation pedigree analysis is $25 with the
proceeds going directly to the Mississippi State University HC
research fund.
Rashmir also has advised stallion and mare owners to include a
provision concerning HC in their breeding contracts in case a foal
is affected.
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JURY AWARDS $1,007,500 IN EQUITROL LAWSUIT
Santa Ana, CA, March 25, 2004 -- The jury in Wrather v. Farnam
Companies returned a verdict of $1,007,500 in favor of plaintiffs
Charlotte Wrather, Christopher Wrather and Lori Araki. The jury found that
Farnam's product EquitrolR, a feed-through fly control product, was
defectively designed (not safe when used in the intended manner) and that
it had caused harm to plaintiffs' Thoroughbred racehorses and Thoroughbred
and Warmblood sport horses. Mr. and Mrs. Wrather are the owners of
Cottonwood Ranch in Los Alamos, California, a Thoroughbred breeding and
training farm. Ms. Araki is manager and trainer at Cottonwood Ranch.
EquitrolR works in the manure to kill fly larvae before they mature.
Its active ingredient is the organophosphate insecticide
tetrachlorvinphos, a cholinesterase inhibito r and neurotoxin which is
also known by the trade name RabonR. Feed-through fly control products
containing RabonR are widely used in beef and dairy cattle and other
livestock industries, as well as in horses.
Farnam has advertised that EquitrolR is designed to pass quickly
through the horse's gastrointestinal tract without being digested, and
that it is safe for all horses including pregnant and lactating mares and
their foals. The Wrathers and Ms. Araki claimed that they fed EquitrolR as
directed, that the organophosphate in it was absorbed into their
horses'systems, and that this caused or exacerbated a variety of health
problems in the horses including reproductive problems and birth defects,
stunted and retarded growth, hyperexcitability and other neurological
dysfunctions, laminitis, immunosuppression evidenced by unusual or
unusually severe infections, low thyroid, diarrhea, colic and more.
Testifying as an expert witness for plaintiffs was Dr. John Madigan,
D.V.M., professor in the Department of Medicine and Epidemiology and chief
of the Equine Section at the Veterinary Medical Teaching Hospital, School
of Veterinary Medicine at the University of California, Davis. Dr. Madigan
last summer conducted a pilot study of the effects of feeding EquitrolR.
In his study (forthcoming in the veterinary literature), the group of test
horses fed EquitrolR experienced a sharp drop in their whole blood
cholinesterase to levels consistent with organophosphate intoxification.
The study also revealed statistically significant differences in behavior
while on EquitrolR as compared with the control group. In a series of
behavioral tests, the horses fed EquitrolR exhibited heightened or
intensified flight response, that is, they were "spookier" or
more easily startled or frightened.
Also testifying for the plaintiffs were Drs. Mark Rick, D.V.M. and Greg
Parks, D.V.M., both of the well known Alamo Pintado Equine Medical Center
in Los Olivos, California; Dr. David Jensen, D.V.M., who practices
privately as San Marcos Equine Practice in Los Alamos, California; and Dr.
Warren Porter, Professor of Environmental Toxicology at the University of
Wisconsin at Madison. The Wrathers and Ms. Araki had also alleged that
Farnam knew at least since 1981 that 10% to 30% of the organophosphate in
EquitrolR was absorbed, so that the advertising and marketing for
EquitrolR contained negligent and intentional misrepresentations and
omissions. The jury did not find that this had been proved by a
preponderance of the evidence.
Charlotte Wrather et al v. Farnam Companies, Inc., United States
District Court for the Central District of California, Santa Ana Civil No.
03-967 JVS(RCx)(March 25, 2004)
For the latest information on horse disease HERDA,
go to:
"Hereditary Disease Research at UC Davis"
http://www.thehorse.com/news.asp?fid=5038
"Inherited Quarter Horse Disease Traces to
Poco Bueno"
http://www.thehorse.com/news.asp?fid=5037
Health Section "The Fatal Flaw",
Part One 3/12/04
http://horsecity.com/stories/031204/hea_herda_KT.shtml
"The Fatal Flaw", Part Two 3/16/04
http://horsecity.com/stories/031604/hea_herda2_KT.shtml
Tips for Reducing Your
Horse's
West Nile Risk
Since first being recognized in
the United States in 1999, West Nile virus (WNV) has posed a serious
threat to horses and humans alike. In the equine population, the
virus is transmitted when a mosquito takes a blood meal from a bird
infected with WMV, then feeds on a horse. While many horses exposed
to WNV experience no signs of illness, the virus can cause inflammation of
the brain and spinal cord. In some cases, especially in older
horses, WNV can be fatal.
For
information about the virus, ask your equine veterinarian for the
"West Nile Virus" brochure, produced by the AAEP in conjunction
with Bayer Animal Health, an AAEP Educational Partner. Additional
information about WNV can be found on the AAEP's horse-heath Web site, www.myHorseMatters.com.
Type West Nile Virus in the search section.
The American Association of
Equine Practitioners, headquartered in Lexington, KY, was founded in 1954
as a non-profit organization dedicated to the health and welfare of the
horse. Currently, AAEP reaches more than 5 million horse owners
through its 6,500 members worldwide and is actively involved in ethics
issues, practice management, research and continuing education in the
equine veterinary profession and horse industry.
Stopping mosquitos
is the key to stopping West Nile virus. Limiting their habitat helps
limit the spread of the virus.
Empty sources of standing water. Mosquitoes lay eggs
in standing water. Here are some tips:
* Every 2-3 days, drain water from
outside pet dishes, garbage cans, buckets, toys, flower pots,
wading pools, pool covers, birdbaths and other objects that can collect
water.
* Discard any old tires and unused containers.
* Clean gutters
* Drill holes in the bottoms of recycling bins.
* Properly treat water in larger pools and home
ponds. Contact your state's department of health for guidelines.
* Mosquitoes like tall grass and weeds. Keep these
cut.
Here's a tip that was given at a recent gardening forum.
Put some water in a white dinner plate and add a couple drops of Lemon
Fresh Joy dish detergent. Set the dish on your porch, patio, or other
outdoor area. Not sure what attracts them, the lemon smell, the white
plate color, or what, but mosquitoes flock to it, and drop dead shortly
after drinking the Lemon Fresh Joy/water mixture, and usually within about
10 feet of the plate. Check this out---it works just super! May seem
trivial, but it may help control mosquitoes around your home.
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